If the blood pressure is too low, the cardiovascular control centre in the medulla oblongata attempts to compensate by causing a/an _______.

Decreased cardiac output

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Increased contractility of the myocardial cells

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Systemic arteriolar dilation

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Decreased heart rate

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Solution

The correct option is B
Increased contractility of the myocardial cells

If the blood pressure is too low, the cardiovascular control centre in the medulla oblongata attempts to compensate by causing an increased contractility of the myocardial cells.

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The cardiac activity could be moderated by the autonomous neural system.
Tick the correct answer.
(a) The parasympathetic system stimulates heart rate and stroke volume
(b) The sympathetic system stimulates heart rate and stroke volume
(c) The parasympathetic system decrease the heart rate but increase stroke volume
(d) The sympathetic system decreases the heart rate but increases stroke volume

If the blood pressure is too low, the cardiovascular control centre in the medulla oblongata attempts to compensate by causing a/an _______.

Q. An increase in blood pressure can cause the release of , a way to control the RAAS system.

Q. Neural signal through sympathetic component of autonomic nervous system (ANS) can

a. Increase the heart beat rate
b. Decrease the heart beat rate
c. Increase the strength of ventricular contraction
d. Decrease the speed of conduction of action potential
e. Increase cardiac output
f. Decrease cardiac output

If the blood pressure is too low, the cardiovascular control centre in the medulla oblongata attempts to compensate by causing a/an _______.

The correct option is B Increased contractility of the myocardial cells If the blood pressure is too low, the cardiovascular control centre in the medulla oblongata attempts to compensate by causing an increased contractility of the myocardial cells.

The correct option is B
Increased contractility of the myocardial cells

If the blood pressure is too low, the cardiovascular control centre in the medulla oblongata attempts to compensate by causing an increased contractility of the myocardial cells.